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Anti-TNFα therapy may lead to blood pressure reductions through improved endothelium-dependent microvascular function in patients with rheumatoid arthritis

  • A Sandoo
  • , V F Panoulas
  • , T E Toms
  • , J P Smith
  • , A Stavropoulos-Kalinoglou
  • , G S Metsios
  • , A Y Gasparyan
  • , D Carroll
  • , J J C S Veldhuijzen van Zanten
  • , G D Kitas
  • Dudley Group NHS, Dudley

Research output: Contribution to journalArticlepeer-review

Original languageEnglish
Pages (from-to)699-702
Number of pages4
JournalJournal of human hypertension
Volume25
Issue number11
DOIs
Publication statusPublished - Nov 2011
Externally publishedYes

Keywords

  • Adalimumab
  • Antibodies, Monoclonal
  • Antibodies, Monoclonal, Humanized
  • Antirheumatic Agents
  • Arthritis, Rheumatoid
  • Blood Pressure
  • Endothelium, Vascular
  • England
  • Etanercept
  • Humans
  • Immunoglobulin G
  • Inflammation Mediators
  • Infliximab
  • Longitudinal Studies
  • Microvessels
  • Receptors, Tumor Necrosis Factor
  • Time Factors
  • Treatment Outcome
  • Tumor Necrosis Factor-alpha
  • Letter
  • Research Support, Non-U.S. Gov't

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